dc.contributor.author |
Rath, Barbara A. |
|
dc.contributor.author |
von Kleist, Max |
|
dc.contributor.author |
Castillo, Maria E. |
|
dc.contributor.author |
Kolevic, Lenka |
|
dc.contributor.author |
Caballero, Patricia |
|
dc.contributor.author |
Soto-Castellares, Giselle |
|
dc.contributor.author |
Amedee, Angela M. |
|
dc.contributor.author |
Robinson, James E. |
|
dc.contributor.author |
Katzenstein, David K. |
|
dc.contributor.author |
Van Dyke, Russell B. |
|
dc.contributor.author |
Oberhelman, Richard A. |
|
dc.date.accessioned |
2022-01-04T20:29:55Z |
|
dc.date.available |
2022-01-04T20:29:55Z |
|
dc.date.issued |
2013 |
|
dc.identifier.uri |
https://hdl.handle.net/20.500.12866/10398 |
|
dc.description.abstract |
Background: The impact of extended use of ART in developing countries has been enormous. A thorough understanding of all factors contributing to the success of antiretroviral therapy is required. The current study aims to investigate the value of cross-sectional drug resistance monitoring using DNA and RNA oligonucleotide ligation assays (OLA) in treatment cohorts in low-resource settings. The study was conducted in the first cohort of children gaining access to structured ART in Peru. Methods: Between 2002–5, 46 eligible children started the standard regimen of AZT, 3TC and NFV Patients had a median age of 5.6 years (range: 0.7-14y), a median viral load of 1.7·105 RNA/ml (range: 2.1·103 – 1.2·106), and a median CD4-count of 232 cells/μL (range: 1–1591). Of these, 20 patients were classified as CDC clinical category C and 31/46 as CDC immune category 3. At the time of cross-sectional analysis in 2005, adherence questionnaires were administered. DNA OLAs and RNA OLAs were performed from fro en PBMC and plasma, RNA genotyping from dried blood spots. Results: During the first year of ART, 44% of children experienced virologic failure, with an additional 9% failing by the end of the second year. Virologic failure was significantly associated with the number of resistance mutations detected by DNA-OLA (p < 0.001) during cross-sectional analysis, but also with low immunologic CDC-scores at baseline (p < 0.001). Children who had been exposed to unsupervised short-term antiretrovirals before starting structured ART showed significantly higher numbers of resistance mutations by DNA-OLA (p = 0.01). Detection of M184V (3TC resistance) by RNA-OLA and DNA-OLA demonstrated a sensitivity of 0.93 and 0.86 and specificity of 0.67 and 0.7, respectively, for the identification of virologic failure. The RT mutations N88D and L90M (NFV resistance) detected by DNA-OLA correlated with virologic failure, whereas mutations at RT position 215 (AZT resistance) were not associated with virologic failure. Conclusions: Advanced immunosuppression at baseline and previous exposures to unsupervised brief cycles of ART significantly impaired treatment outcomes at a time when structured ART was finally introduced in his cohort. Brief maternal exposures to with AZT +/− NVP for the prevention of mother-to-child transmission did not affect treatment outcomes in this group of children. DNA-OLA from frozen PBMC provided a highly specific tool to detect archived drug resistance. RNA consensus genotyping from dried blood spots and RNA-OLA from plasma consistently detected drug resistance mutations, but merely in association with virologic failure. |
en_US |
dc.language.iso |
eng |
|
dc.publisher |
BioMed Central |
|
dc.relation.ispartofseries |
BMC Infectious Diseases |
|
dc.rights |
info:eu-repo/semantics/restrictedAccess |
|
dc.rights.uri |
https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es |
|
dc.subject |
Cross-Sectional Studies |
en_US |
dc.subject |
human |
en_US |
dc.subject |
Peru |
en_US |
dc.subject |
questionnaire |
en_US |
dc.subject |
sensitivity and specificity |
en_US |
dc.subject |
HIV Infections |
en_US |
dc.subject |
Human immunodeficiency virus infection |
en_US |
dc.subject |
treatment outcome |
en_US |
dc.subject |
human cell |
en_US |
dc.subject |
antiretrovirus agent |
en_US |
dc.subject |
cohort analysis |
en_US |
dc.subject |
Human immunodeficiency virus infected patient |
en_US |
dc.subject |
CD4 lymphocyte count |
en_US |
dc.subject |
genotype |
en_US |
dc.subject |
article |
en_US |
dc.subject |
antiviral therapy |
en_US |
dc.subject |
CD4 Lymphocyte Count |
en_US |
dc.subject |
HIV-1 |
en_US |
dc.subject |
clinical article |
en_US |
dc.subject |
Antiretroviral Therapy, Highly Active |
en_US |
dc.subject |
genetic analysis |
en_US |
dc.subject |
genetic association |
en_US |
dc.subject |
drug exposure |
en_US |
dc.subject |
treatment duration |
en_US |
dc.subject |
immunosuppressive treatment |
en_US |
dc.subject |
drug treatment failure |
en_US |
dc.subject |
virus RNA |
en_US |
dc.subject |
vertical transmission |
en_US |
dc.subject |
RNA |
en_US |
dc.subject |
peripheral blood mononuclear cell |
en_US |
dc.subject |
health care access |
en_US |
dc.subject |
DNA |
en_US |
dc.subject |
disease classification |
en_US |
dc.subject |
Treatment Failure |
en_US |
dc.subject |
Mutation |
en_US |
dc.subject |
dried blood spot testing |
en_US |
dc.subject |
virus load |
en_US |
dc.subject |
Disease Progression |
en_US |
dc.subject |
Viral Load |
en_US |
dc.subject |
antiviral resistance |
en_US |
dc.subject |
blood analysis |
en_US |
dc.subject |
consensus sequence |
en_US |
dc.subject |
drug monitoring |
en_US |
dc.subject |
Drug Resistance, Viral |
en_US |
dc.subject |
Genes, Viral |
en_US |
dc.subject |
lamivudine |
en_US |
dc.subject |
nelfinavir |
en_US |
dc.subject |
oligonucleotide ligation assay |
en_US |
dc.subject |
virus mutation |
en_US |
dc.subject |
zidovudine |
en_US |
dc.title |
Antiviral Resistance and Correlates of Virologic Failure in the first Cohort of HIV-Infected Children Gaining Access to Structured Antiretroviral Therapy in Lima, Peru: A Cross-Sectional Analysis |
en_US |
dc.type |
info:eu-repo/semantics/article |
|
dc.identifier.doi |
https://doi.org/10.1186/1471-2334-13-1 |
|
dc.subject.ocde |
https://purl.org/pe-repo/ocde/ford#3.03.08 |
|
dc.relation.issn |
1471-2334 |
|