dc.contributor.author |
Rivera, Fulton P. |
|
dc.contributor.author |
Medina, Anicia M. |
|
dc.contributor.author |
Bezada, Sandra |
|
dc.contributor.author |
Valencia, Roberto |
|
dc.contributor.author |
Bernal, María |
|
dc.contributor.author |
Meza, Rina |
|
dc.contributor.author |
Maves, Ryan C. |
|
dc.contributor.author |
Ochoa Woodell, Theresa Jean |
|
dc.date.accessioned |
2022-01-04T20:31:49Z |
|
dc.date.available |
2022-01-04T20:31:49Z |
|
dc.date.issued |
2013 |
|
dc.identifier.uri |
https://hdl.handle.net/20.500.12866/10697 |
|
dc.description.abstract |
Secretory diarrhea caused by cholera toxin (CT) is initiated by binding of CT’s B subunit (CTB) to GM1-ganglioside on the surface of intestinal cells. Lactoferrin, a breast milk glycoprotein, has shown protective effect against several enteropathogens. The aims of this study were to determine the effect of bovine-lactoferrin (bLF) on CT-induced intestinal fluid accumulation in mice, and the interaction between bLF and CT/CTB with the GM1-ganglioside receptor. Fluid accumulation induced by CT was evaluated in the mouse ileal loop model using 56 BALB/c mice, with and without bLF added before, after or at the same time of CT administration. The effect of bLF in the interaction of CT and CTB with GM1-ganglioside was evaluated by a GM1-enzyme-linked immunosorbent assay. bLF decreased CT-induced fluid accumulation in the ileal loop of mice. The greatest effect was when bLF was added before CT (median, 0.066 vs. 0.166 g/cm, with and without bLF respectively, p<0.01). We conclude that bLF decreases binding of CT and CTB to GM1-ganglioside, suggesting that bLF suppresses CT-induced fluid accumulation by blocking the binding of CTB to GM1-ganglioside. bLF may be effective as adjunctive therapy for treatment of cholera diarrhea. |
en_US |
dc.language.iso |
eng |
|
dc.publisher |
Public Library of Science |
|
dc.relation.ispartofseries |
PLoS ONE |
|
dc.rights |
info:eu-repo/semantics/restrictedAccess |
|
dc.rights.uri |
https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es |
|
dc.subject |
Animals |
en_US |
dc.subject |
Mice |
en_US |
dc.subject |
Cattle |
en_US |
dc.subject |
Dose-Response Relationship, Drug |
en_US |
dc.subject |
Chlorides |
en_US |
dc.subject |
Cholera Toxin |
en_US |
dc.subject |
Enterotoxins |
en_US |
dc.subject |
Escherichia coli |
en_US |
dc.subject |
Ferric Compounds |
en_US |
dc.subject |
G(M1) Ganglioside |
en_US |
dc.subject |
Gangliosides |
en_US |
dc.subject |
Intestinal Mucosa |
en_US |
dc.subject |
Intestines |
en_US |
dc.subject |
Lactoferrin |
en_US |
dc.subject |
Protein Binding |
en_US |
dc.subject |
Receptors Cell Surface |
en_US |
dc.title |
Bovine Lactoferrin Decreases Cholera-Toxin-Induced Intestinal Fluid Accumulation in Mice by Ganglioside Interaction |
en_US |
dc.type |
info:eu-repo/semantics/article |
|
dc.identifier.doi |
https://doi.org/10.1371/journal.pone.0059253 |
|
dc.subject.ocde |
https://purl.org/pe-repo/ocde/ford#1.06.11 |
|
dc.relation.issn |
1932-6203 |
|