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dc.contributor.author | Mendoza, A. | |
dc.contributor.author | Pérez-Silanes, S. | |
dc.contributor.author | Quiliano, M. | |
dc.contributor.author | Pabón, A. | |
dc.contributor.author | Galiano, S. | |
dc.contributor.author | González, G. | |
dc.contributor.author | Garavito, G. | |
dc.contributor.author | Zimic-Peralta, Mirko Juan | |
dc.contributor.author | Vaisberg Wollach, Abraham Jaime | |
dc.contributor.author | Aldana, I. | |
dc.contributor.author | Monge, A. | |
dc.contributor.author | Deharo, E. | |
dc.date.accessioned | 2022-01-18T19:26:46Z | |
dc.date.available | 2022-01-18T19:26:46Z | |
dc.date.issued | 2011 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12866/10854 | |
dc.description.abstract | Piperazine and pyrrolidine derivatives were synthesised and evaluated for their capacity to inhibit the growth of Plasmodium falciparum chloroquine-resistant (FCR-3) strain in culture. The combined presence of a hydroxyl group, a propane chain and a fluor were shown to be crucial for the antiplasmodial activity. Five compounds of the aryl-alcohol series inhibited 50% of parasite growth at doses ≤10μM. The most active compound 1-(4-fluoronaphthyl)-3-[4-(4-nitro-2-trifluoromethylphenyl)piperazin-1-yl] propan-1-ol was almost 20-40 times more active on P. falciparum (IC50: 0.5μM) than on tumorogenic and non-tumorogenic cells. In vivo it has a very weak effect; inhibiting 35% of parasite growth only, at 10mg/kg/day against Plasmodium berghei infected mice without any impact on survival time. In silico molecular docking study and molecular electrostatic potential calculation revealed that this compound bound to the active site of Plasmodium plasmepsin II enzyme. | en_US |
dc.language.iso | eng | |
dc.publisher | Elsevier | |
dc.relation.ispartofseries | Experimental Parasitology | |
dc.rights | info:eu-repo/semantics/restrictedAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es | |
dc.subject | Humans | en_US |
dc.subject | Controlled Study | en_US |
dc.subject | Unclassified Drug | en_US |
dc.subject | Plasmodium Falciparum | en_US |
dc.subject | Antimalarial Agent | en_US |
dc.subject | Chloroquine | en_US |
dc.subject | Cell Line | en_US |
dc.subject | Doxycycline | en_US |
dc.subject | Antimalarials | en_US |
dc.subject | Host Parasite Interaction | en_US |
dc.subject | Parasite Development | en_US |
dc.subject | Protozoal DNA | en_US |
dc.subject | Monotherapy | en_US |
dc.subject | Developmental Stage | en_US |
dc.subject | Plant Extracts | en_US |
dc.subject | Cell Proliferation | en_US |
dc.subject | Antimalarial Activity | en_US |
dc.subject | Combination Chemotherapy | en_US |
dc.subject | Trophozoite | en_US |
dc.subject | Artemether | en_US |
dc.subject | Inhibitory Concentration 50 | en_US |
dc.subject | Amodiaquine | en_US |
dc.subject | Apoptosis | en_US |
dc.subject | Atovaquone | en_US |
dc.subject | Bruceantin | en_US |
dc.subject | Brusatol | en_US |
dc.subject | Cell Death | en_US |
dc.subject | Cell Membrane Permeability | en_US |
dc.subject | DNA Replication | en_US |
dc.subject | Erythrocyte Membrane | en_US |
dc.subject | Erythrocytes | en_US |
dc.subject | Halofantrine | en_US |
dc.subject | Heme | en_US |
dc.subject | Lymphoblast | en_US |
dc.subject | Mefloquine | en_US |
dc.subject | Plant Medicinal Product | en_US |
dc.subject | Plasmodium Yoelii | en_US |
dc.subject | Quassia Amara | en_US |
dc.subject | Quassinoid | en_US |
dc.subject | Simalikalactone D | en_US |
dc.subject | Simaroubaceae | en_US |
dc.title | Aryl piperazine and pyrrolidine as antimalarial agents. Synthesis and investigation of structure-activity relationships | en_US |
dc.type | info:eu-repo/semantics/article | |
dc.identifier.doi | https://doi.org/10.1016/j.exppara.2011.02.025 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#3.03.07 | |
dc.relation.issn | 1090-2449 |
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