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dc.contributor.author | Micheal, Shazia | |
dc.contributor.author | Topouzis, Fotis | |
dc.contributor.author | Craig, Jamie E. | |
dc.contributor.author | Dubina, Michael | |
dc.contributor.author | Sundaresan, Periasamy | |
dc.contributor.author | Stefansson, Kari | |
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dc.date.accessioned | 2019-01-25T15:18:35Z | |
dc.date.available | 2019-01-25T15:18:35Z | |
dc.date.issued | 2017 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12866/4663 | |
dc.description.abstract | Exfoliation syndrome (XFS) is the most common known risk factor for secondary glaucoma and a major cause of blindness worldwide. Variants in two genes, LOXL1 and CACNA1A, have previously been associated with XFS. To further elucidate the genetic basis of XFS, we collected a global sample of XFS cases to refine the association at LOXL1, which previously showed inconsistent results across populations, and to identify new variants associated with XFS. We identified a rare protective allele at LOXL1 (p.Phe407, odds ratio (OR) = 25, P = 2.9 x 10(-14)) through deep resequencing of XFS cases and controls from nine countries. A genome-wide association study (GWAS) of XFS cases and controls from 24 countries followed by replication in 18 countries identified seven genome-wide significant loci (P < 5 x 10(-8)). We identified association signals at 13q12 (POMP), 11q23.3 (TMEM136), 6p21 (AGPAT1), 3p24 (RBMS3) and 5q23 (near SEMA6A). These findings provide biological insights into the pathology of XFS and highlight a potential role for naturally occurring rare LOXL1 variants in disease biology. | en_US |
dc.language.iso | eng | |
dc.publisher | Springer Nature | |
dc.relation.ispartofseries | Nature Genetics | |
dc.rights | info:eu-repo/semantics/restrictedAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es | |
dc.subject | Female | en_US |
dc.subject | Humans | en_US |
dc.subject | Male | en_US |
dc.subject | Aged, 80 and over | en_US |
dc.subject | Alleles | en_US |
dc.subject | Haplotypes | en_US |
dc.subject | Genome-Wide Association Study | en_US |
dc.subject | Mutation, Missense | en_US |
dc.subject | Point Mutation | en_US |
dc.subject | Amino Acid Oxidoreductases/genetics/physiology | en_US |
dc.subject | Amino Acid Substitution | en_US |
dc.subject | Asian Continental Ancestry Group/genetics | en_US |
dc.subject | Calcium Channels/genetics | en_US |
dc.subject | Cell Adhesion | en_US |
dc.subject | Exfoliation Syndrome/ethnology/genetics | en_US |
dc.subject | Extracellular Matrix/metabolism | en_US |
dc.subject | Eye/metabolism | en_US |
dc.subject | Gene Expression Profiling | en_US |
dc.subject | Genetic Predisposition to Disease | en_US |
dc.subject | Molecular Chaperones/biosynthesis/genetics | en_US |
dc.subject | RNA, Messenger/biosynthesis | en_US |
dc.subject | Spheroids, Cellular | en_US |
dc.title | Genetic association study of exfoliation syndrome identifies a protective rare variant at LOXL1 and five new susceptibility loci | en_US |
dc.type | info:eu-repo/semantics/article | |
dc.identifier.doi | https://doi.org/10.1038/ng.3875 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#1.06.07 | |
dc.relation.issn | 1546-1718 |
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