dc.contributor.author |
Rosado, Jason |
|
dc.contributor.author |
Morales, Sandra |
|
dc.contributor.author |
Lopez, Giovanni |
|
dc.contributor.author |
Clark, Daniel |
|
dc.contributor.author |
Verdonck, Kristien |
|
dc.contributor.author |
Gotuzzo Herencia, José Eduardo |
|
dc.contributor.author |
Van Camp, Guy |
|
dc.contributor.author |
Talledo Albujar, Michael John |
|
dc.date.accessioned |
2019-01-25T16:03:21Z |
|
dc.date.available |
2019-01-25T16:03:21Z |
|
dc.date.issued |
2016 |
|
dc.identifier.uri |
https://hdl.handle.net/20.500.12866/4776 |
|
dc.description.abstract |
Human T‐lymphotropic virus 1 (HTLV‐1) is the etiologic agent of the HTLV‐1‐associated myelopathy/tropical spastic paraparesis (HAM/TSP). Apoptosis is a mechanism of defense elicited by many triggers, including cross‐linking of the FAS receptor expressed in viruses‐infected cells, and the ligand FASL presented by T‐cytotoxic cells. As HAM/TSP has been associated with high levels of proviral load (PVL), we hypothesized that certain genotypes of single‐nucleotide polymorphisms (SNPs) associated with a decreased protein expression of FAS and FASL could be risk factors for this disease. Three SNPs: FAS‐670A/G (rs1800682), FAS‐1377G/A (rs2234767), and FASL‐844C/T (rs763110) were analyzed in 73 HAM/TSP patients and 143 HTLV‐1 asymptomatic carriers. Ancestry informative markers were used to adjust for ethnicity through a principal component analysis. Gender, age, PVL, and the first three principal components were used as covariates. The FAS/FASL genotype distribution was not associated with HAM/TSP presence (P–> 0.05). The FAS‐670 AA genotype was associated with high PVL in comparison to FAS‐670 GG in HAM/TSP patients (P = 0.015), while in asymptomatic carriers low levels of PVL were observed (P > 0.05). Our findings suggest that rs1800682, rs2234767, and rs763110 genotypes are not associated with the presence of HAM/TSP, but that the FAS‐670 AA genotype can promote higher PVL values in HAM/TSP patients. |
en_US |
dc.language.iso |
eng |
|
dc.publisher |
Wiley |
|
dc.relation.ispartofseries |
Journal of Medical Virology |
|
dc.rights |
info:eu-repo/semantics/restrictedAccess |
|
dc.rights.uri |
https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es |
|
dc.subject |
antigens, CD95 |
en_US |
dc.subject |
Genotype |
en_US |
dc.subject |
HTLV-I-associated myelopathy-tropical spastic paraparesis |
en_US |
dc.subject |
Human T-lymphotropic virus 1 |
en_US |
dc.subject |
polymorphism, single nucleotide |
en_US |
dc.subject |
Polymorphism, Single Nucleotide |
en_US |
dc.subject |
proviral load |
en_US |
dc.subject |
Viral Load |
en_US |
dc.subject |
Cohort Studies |
en_US |
dc.subject |
fas Receptor/genetics |
en_US |
dc.subject |
Female |
en_US |
dc.subject |
HTLV-I Infections/virology |
en_US |
dc.subject |
Humans |
en_US |
dc.subject |
Male |
en_US |
dc.subject |
Middle Aged |
en_US |
dc.subject |
Proviruses/genetics |
en_US |
dc.title |
The FAS-670 AA genotype is associated with high proviral load in peruvian HAM/TSP patients |
en_US |
dc.type |
info:eu-repo/semantics/article |
|
dc.identifier.doi |
https://doi.org/10.1002/jmv.24681 |
|
dc.subject.ocde |
https://purl.org/pe-repo/ocde/ford#1.06.02 |
|
dc.relation.issn |
1096-9071 |
|