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dc.contributor.author | Rosado, Jason | |
dc.contributor.author | Morales, Sandra | |
dc.contributor.author | Lopez, Giovanni | |
dc.contributor.author | Clark, Daniel | |
dc.contributor.author | Verdonck, Kristien | |
dc.contributor.author | Gotuzzo Herencia, José Eduardo | |
dc.contributor.author | Van Camp, Guy | |
dc.contributor.author | Talledo Albujar, Michael John | |
dc.date.accessioned | 2019-01-25T16:03:21Z | |
dc.date.available | 2019-01-25T16:03:21Z | |
dc.date.issued | 2016 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12866/4776 | |
dc.description.abstract | Human T‐lymphotropic virus 1 (HTLV‐1) is the etiologic agent of the HTLV‐1‐associated myelopathy/tropical spastic paraparesis (HAM/TSP). Apoptosis is a mechanism of defense elicited by many triggers, including cross‐linking of the FAS receptor expressed in viruses‐infected cells, and the ligand FASL presented by T‐cytotoxic cells. As HAM/TSP has been associated with high levels of proviral load (PVL), we hypothesized that certain genotypes of single‐nucleotide polymorphisms (SNPs) associated with a decreased protein expression of FAS and FASL could be risk factors for this disease. Three SNPs: FAS‐670A/G (rs1800682), FAS‐1377G/A (rs2234767), and FASL‐844C/T (rs763110) were analyzed in 73 HAM/TSP patients and 143 HTLV‐1 asymptomatic carriers. Ancestry informative markers were used to adjust for ethnicity through a principal component analysis. Gender, age, PVL, and the first three principal components were used as covariates. The FAS/FASL genotype distribution was not associated with HAM/TSP presence (P–> 0.05). The FAS‐670 AA genotype was associated with high PVL in comparison to FAS‐670 GG in HAM/TSP patients (P = 0.015), while in asymptomatic carriers low levels of PVL were observed (P > 0.05). Our findings suggest that rs1800682, rs2234767, and rs763110 genotypes are not associated with the presence of HAM/TSP, but that the FAS‐670 AA genotype can promote higher PVL values in HAM/TSP patients. | en_US |
dc.language.iso | eng | |
dc.publisher | Wiley | |
dc.relation.ispartofseries | Journal of Medical Virology | |
dc.rights | info:eu-repo/semantics/restrictedAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es | |
dc.subject | antigens, CD95 | en_US |
dc.subject | Genotype | en_US |
dc.subject | HTLV-I-associated myelopathy-tropical spastic paraparesis | en_US |
dc.subject | Human T-lymphotropic virus 1 | en_US |
dc.subject | polymorphism, single nucleotide | en_US |
dc.subject | Polymorphism, Single Nucleotide | en_US |
dc.subject | proviral load | en_US |
dc.subject | Viral Load | en_US |
dc.subject | Cohort Studies | en_US |
dc.subject | fas Receptor/genetics | en_US |
dc.subject | Female | en_US |
dc.subject | HTLV-I Infections/virology | en_US |
dc.subject | Humans | en_US |
dc.subject | Male | en_US |
dc.subject | Middle Aged | en_US |
dc.subject | Proviruses/genetics | en_US |
dc.title | The FAS-670 AA genotype is associated with high proviral load in peruvian HAM/TSP patients | en_US |
dc.type | info:eu-repo/semantics/article | |
dc.identifier.doi | https://doi.org/10.1002/jmv.24681 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#1.06.02 | |
dc.relation.issn | 1096-9071 |
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