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Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E

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dc.contributor.author Diaz, Victor
dc.contributor.author Peinado, Ana B.
dc.contributor.author Barba-Moreno, Laura
dc.contributor.author Altamura, Sandro
dc.contributor.author Butragueno, Javier
dc.contributor.author Gonzalez-Gross, Marcela
dc.contributor.author Alteheld, Birgit
dc.contributor.author Stehle, Peter
dc.contributor.author Zapico, Augusto G.
dc.contributor.author Muckenthaler, Martina U.
dc.contributor.author Gassmann, Max
dc.date.accessioned 2019-02-06T14:53:08Z
dc.date.available 2019-02-06T14:53:08Z
dc.date.issued 2015
dc.identifier.uri https://hdl.handle.net/20.500.12866/5378
dc.description.abstract Iron deficiency among endurance athletes is of major concern for coaches, physicians, and nutritionists. Recently, it has been observed that hepcidin, the master regulator of iron metabolism, was upregulated after exercise and was found to be related to interleukin-6 (IL-6) elevation. In this study performed on noniron deficient and well-trained runners, we observed that hepcidin concentrations remain elevated in response to inflammatory and iron signals despite a 28-days supplementation period with vitamins C (500 mg/day) and E (400 IU/day). en_US
dc.language.iso eng
dc.publisher Wiley
dc.relation.ispartof urn:issn:2051-817X
dc.rights info:eu-repo/semantics/restrictedAccess
dc.rights.uri https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es
dc.subject performance en_US
dc.subject interleukin-6 en_US
dc.subject Endurance en_US
dc.subject iron deficiency en_US
dc.subject vitamins en_US
dc.title Elevated hepcidin serum level in response to inflammatory and iron signals in exercising athletes is independent of moderate supplementation with vitamin C and E en_US
dc.type info:eu-repo/semantics/article
dc.identifier.doi https://doi.org/10.14814/phy2.12475
dc.subject.ocde https://purl.org/pe-repo/ocde/ford#3.02.00 es_PE
dc.subject.ocde https://purl.org/pe-repo/ocde/ford#3.01.08


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