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dc.contributor.author Ugarte Gil, Cesar Augusto
dc.contributor.author Pearson, F.
dc.contributor.author Moore, David Alexander James
dc.contributor.author Critchley, J.
dc.contributor.author van Crevel, R.
dc.date.accessioned 2019-08-08T15:23:45Z
dc.date.available 2019-08-08T15:23:45Z
dc.date.issued 2019
dc.identifier.uri https://hdl.handle.net/20.500.12866/7133
dc.description.abstract We thank Drs Yates and Barr for their valuable comments. In our article [1], we were not attempting to assess the extent to which dysglycemia is causally associated with tuberculosis (TB). A number of prospective studies exist, among a body of evidence, supporting probabilistic causation between diabetes mellitus (DM) and TB [2, 3]. Rather, the aim of our study was to identify age-adjusted prevalence and clinical characteristics of DM and intermediate hyperglycemia among those with newly diagnosed TB across 4 TB-endemic settings. In our South African population, the prevalence of DM (10.9% [95% confidence interval, 7%–14.9%]) was the... en_US
dc.language.iso eng
dc.publisher Oxford University Press
dc.relation.ispartofseries Clinical Infectious Diseases
dc.rights info:eu-repo/semantics/restrictedAccess
dc.rights.uri https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es
dc.subject tuberculosis en_US
dc.subject dysglycemia en_US
dc.subject diabetes mellitus en_US
dc.subject associated factors en_US
dc.title Reply to Yates and Barr en_US
dc.type info:eu-repo/semantics/article
dc.identifier.doi https://doi.org/10.1093/cid/ciz434
dc.subject.ocde https://purl.org/pe-repo/ocde/ford#3.03.08
dc.relation.issn 1537-6591


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