Reply to Yates and Barr

Ugarte Gil, Cesar Augusto; Critchley, J.; Moore, David Alexander James; Pearson, F.; van Crevel, R.

dc.contributor.author	Ugarte Gil, Cesar Augusto
dc.contributor.author	Pearson, F.
dc.contributor.author	Moore, David Alexander James
dc.contributor.author	Critchley, J.
dc.contributor.author	van Crevel, R.
dc.date.accessioned	2019-08-08T15:23:45Z
dc.date.available	2019-08-08T15:23:45Z
dc.date.issued	2019
dc.identifier.uri	https://hdl.handle.net/20.500.12866/7133
dc.description.abstract	We thank Drs Yates and Barr for their valuable comments. In our article [1], we were not attempting to assess the extent to which dysglycemia is causally associated with tuberculosis (TB). A number of prospective studies exist, among a body of evidence, supporting probabilistic causation between diabetes mellitus (DM) and TB [2, 3]. Rather, the aim of our study was to identify age-adjusted prevalence and clinical characteristics of DM and intermediate hyperglycemia among those with newly diagnosed TB across 4 TB-endemic settings. In our South African population, the prevalence of DM (10.9% [95% confidence interval, 7%–14.9%]) was the...	en_US
dc.language.iso	eng
dc.publisher	Oxford University Press
dc.relation.ispartofseries	Clinical Infectious Diseases
dc.rights	info:eu-repo/semantics/restrictedAccess
dc.rights.uri	https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es
dc.subject	tuberculosis	en_US
dc.subject	dysglycemia	en_US
dc.subject	diabetes mellitus	en_US
dc.subject	associated factors	en_US
dc.title	Reply to Yates and Barr	en_US
dc.type	info:eu-repo/semantics/article
dc.identifier.doi	https://doi.org/10.1093/cid/ciz434
dc.subject.ocde	https://purl.org/pe-repo/ocde/ford#3.03.08
dc.relation.issn	1537-6591