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dc.contributor.author | Siques, Patricia | |
dc.contributor.author | Brito, Julio | |
dc.contributor.author | Naveas, Nelson | |
dc.contributor.author | Pulido, Ruth | |
dc.contributor.author | De La Cruz, Juan José | |
dc.contributor.author | Mamani, Maribel | |
dc.contributor.author | León-Velarde, Fabiola | |
dc.date.accessioned | 2020-06-10T18:11:30Z | |
dc.date.available | 2020-06-10T18:11:30Z | |
dc.date.issued | 2014 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12866/7957 | |
dc.description.abstract | Plasma and liver lipid profiles in rats exposed to chronic hypobaric hypoxia: Changes in metabolic pathways. High Alt Med Biol 15:388-395, 2014. - Lipid metabolism under chronic hypoxia (CH) has not received equal attention as intermittent hypoxia (IH). To determine the CH-induced changes in plasma and liver, as well as the mRNA and protein expression of two key enzymes in the triglyceride and cholesterol biosynthesis pathways, SREBP-1 (HMG-CoA reductase) and SREBP-2 (SCD-1), we exposed adult male Wistar rats to CH (4600 m; n=15) for 30 days compared to normoxic rats (n=15). The CH rats exhibited weight loss (p<0.001), higher hematocrit (%), and higher hemoglobin (g/dL) (p<0.01). In the plasma of CH rats, total cholesterol and LDL-cholesterol increased at day 15. VLDL-cholesterol and triglycerides (p<0.01) greatly increased (35%), while HDL-cholesterol decreased (p<0.01). Triglycerides and VLDL-cholesterol remained elevated by 28% at day 30 (p<0.01). Hepatic triglycerides increased two-fold, while total cholesterol increased by 51% (p<0.001; p<0.05). Upregulation of SCD-1 mRNA and protein was observed in the CH rats (p<0.01); however, no differences were observed in HMG-CoA reductase mRNA or protein expression in both groups. In conclusion, CH, like IH, alters lipid profiles by increasing triglycerides in the plasma and liver and upregulating triglyceride biosynthesis without affecting the cholesterol biosynthetic pathway. Additional involved mechanisms require further study because of the importance of lipids in cardiovascular risk. | en_US |
dc.language.iso | eng | |
dc.publisher | Mary Ann Liebert | |
dc.relation.ispartofseries | High Altitude Medicine and Biology | |
dc.rights | info:eu-repo/semantics/restrictedAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es | |
dc.subject | acyl coenzyme A desaturase | en_US |
dc.subject | adult | en_US |
dc.subject | animal | en_US |
dc.subject | animal experiment | en_US |
dc.subject | animal model | en_US |
dc.subject | animal tissue | en_US |
dc.subject | Animals | en_US |
dc.subject | anoxia | en_US |
dc.subject | Anoxia | en_US |
dc.subject | Article | en_US |
dc.subject | biological marker | en_US |
dc.subject | Biological Markers | en_US |
dc.subject | blood lipids | en_US |
dc.subject | Blotting, Western | en_US |
dc.subject | cholesterol | en_US |
dc.subject | Cholesterol | en_US |
dc.subject | cholesterol blood level | en_US |
dc.subject | cholesterol liver level | en_US |
dc.subject | chronic disease | en_US |
dc.subject | Chronic Disease | en_US |
dc.subject | chronic hypobaric hypoxia | en_US |
dc.subject | chronic hypoxia | en_US |
dc.subject | controlled study | en_US |
dc.subject | enzymology | en_US |
dc.subject | evaluation study | en_US |
dc.subject | hematocrit | en_US |
dc.subject | Hematocrit | en_US |
dc.subject | hemoglobin | en_US |
dc.subject | hemoglobin blood level | en_US |
dc.subject | Hemoglobins | en_US |
dc.subject | high altitude | en_US |
dc.subject | high density lipoprotein cholesterol | en_US |
dc.subject | HMG-CoA reductase | en_US |
dc.subject | Hydroxymethylglutaryl CoA Reductases | en_US |
dc.subject | hydroxymethylglutaryl coenzyme A reductase | en_US |
dc.subject | hypobarism | en_US |
dc.subject | hypoxia | en_US |
dc.subject | lipid | en_US |
dc.subject | lipid liver level | en_US |
dc.subject | lipid metabolism | en_US |
dc.subject | liver | en_US |
dc.subject | Liver | en_US |
dc.subject | low density lipoprotein cholesterol | en_US |
dc.subject | male | en_US |
dc.subject | Male | en_US |
dc.subject | messenger RNA | en_US |
dc.subject | metabolism | en_US |
dc.subject | nonhuman | en_US |
dc.subject | protein expression | en_US |
dc.subject | rat | en_US |
dc.subject | Rats | en_US |
dc.subject | Rats, Wistar | en_US |
dc.subject | Reverse Transcriptase Polymerase Chain Reaction | en_US |
dc.subject | reverse transcription polymerase chain reaction | en_US |
dc.subject | SCD-1 | en_US |
dc.subject | Stearoyl-CoA Desaturase | en_US |
dc.subject | stearoyl-CoA desaturase SCD-1, rat | en_US |
dc.subject | sterol regulatory element binding protein 1 | en_US |
dc.subject | sterol regulatory element binding protein 2 | en_US |
dc.subject | triacylglycerol | en_US |
dc.subject | triacylglycerol blood level | en_US |
dc.subject | Triglycerides | en_US |
dc.subject | Up-Regulation | en_US |
dc.subject | upregulation | en_US |
dc.subject | very low density lipoprotein cholesterol | en_US |
dc.subject | weight reduction | en_US |
dc.subject | Western blotting | en_US |
dc.subject | Wistar rat | en_US |
dc.title | Plasma and liver lipid profiles in rats exposed to chronic hypobaric hypoxia: Changes in metabolic pathways | en_US |
dc.type | info:eu-repo/semantics/article | |
dc.identifier.doi | https://doi.org/10.1089/ham.2013.1134 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#3.01.08 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#3.03.11 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#3.03.05 | |
dc.relation.issn | 1557-8682 |
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