dc.contributor.author | Moens, B. | |
dc.contributor.author | Pannecouque, C. | |
dc.contributor.author | Lápez, G. | |
dc.contributor.author | Talledo Albujar, Michael John | |
dc.contributor.author | Gotuzzo Herencia, José Eduardo | |
dc.contributor.author | Khouri, R. | |
dc.contributor.author | Bittencourt, A. | |
dc.contributor.author | Farré, L. | |
dc.contributor.author | Galvão-Castro, B. | |
dc.contributor.author | Vandamme, A.-M. | |
dc.contributor.author | Van Weyenbergh, J. | |
dc.date.accessioned | 2022-01-18T19:34:36Z | |
dc.date.available | 2022-01-18T19:34:36Z | |
dc.date.issued | 2012 | |
dc.identifier.uri | https://hdl.handle.net/20.500.12866/11045 | |
dc.description.abstract | Background: IFN- contributes extensively to host immune response upon viral infection through antiviral, pro-apoptotic, antiproliferative and immunomodulatory activities. Although extensively documented in various types of human cancers and viral infections, controversy exists in the exact mechanism of action of IFN- in human immunodeficiency virus type 1 (HIV-1) and human T-lymphotropic virus type 1 (HTLV-1) retroviral infections. Results: IFN- displayed strong anti-HIV-1 effects in HIV-1/HTLV-1 co-infected MT-4 cells in vitro, demonstrated by the dose-dependent inhibition of the HIV-1-induced cytopathic effect (IC50=83.5IU/ml, p<0.0001) and p24 levels in cell-free supernatant (IC50=1.2IU/ml, p<0.0001). In contrast, IFN- treatment did not affect cell viability or HTLV-1 viral mRNA levels in HTLV-1 mono-infected cell lines, based on flow cytometry and nCounter analysis, respectively. However, we were able to confirm the previously described post-transcriptional inhibition of HTLV-1 p19 secretion by IFN- in cell lines (p=0.0045), and extend this finding to primary Adult T cell Leukemia patient samples (p=0.031). In addition, through microarray and nCounter analysis, we performed the first genome-wide simultaneous quantification of complete human and retroviral transciptomes, demonstrating significant transcriptional activation of interferon-stimulated genes without concomitant decrease of HTLV-1 mRNA levels. Conclusions: Taken together, our results indicate that both the absence of in vitro antiproliferative and pro-apoptotic activity as well as the modest post-transcriptional antiviral activity of IFN- against HTLV-1, were not due to a cell-intrinsic defect in IFN- signalisation, but rather represents a retrovirus-specific phenomenon, considering the strong HIV-1 inhibition in co-infected cells. | en_US |
dc.language.iso | eng | |
dc.publisher | BioMed Central | |
dc.relation.ispartofseries | Virology Journal | |
dc.rights | info:eu-repo/semantics/restrictedAccess | |
dc.rights.uri | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.es | |
dc.subject | Humans | en_US |
dc.subject | controlled study | en_US |
dc.subject | in vitro study | en_US |
dc.subject | nucleotide sequence | en_US |
dc.subject | RNA | en_US |
dc.subject | flow cytometry | en_US |
dc.subject | Human T cell leukemia virus 1 | en_US |
dc.subject | Human T-lymphotropic virus 1 | en_US |
dc.subject | human cell | en_US |
dc.subject | messenger RNA | en_US |
dc.subject | Signal Transduction | en_US |
dc.subject | protein secretion | en_US |
dc.subject | cell viability | en_US |
dc.subject | concentration response | en_US |
dc.subject | IC 50 | en_US |
dc.subject | HIV-1 | en_US |
dc.subject | virus RNA | en_US |
dc.subject | alpha interferon | en_US |
dc.subject | CD4+ T lymphocyte | en_US |
dc.subject | Gene Expression Profiling | en_US |
dc.subject | HTLV-1 | en_US |
dc.subject | Gag protein | en_US |
dc.subject | Cell Survival | en_US |
dc.subject | CD4-Positive T-Lymphocytes | en_US |
dc.subject | human genome | en_US |
dc.subject | transcriptome | en_US |
dc.subject | Interferon-alpha | en_US |
dc.subject | microarray analysis | en_US |
dc.subject | Human immunodeficiency virus 1 | en_US |
dc.subject | Antiviral activity | en_US |
dc.subject | cell level | en_US |
dc.subject | gene activation | en_US |
dc.subject | IFN- signaling | en_US |
dc.subject | intracellular signaling | en_US |
dc.subject | leukemia cell | en_US |
dc.subject | protein p19 | en_US |
dc.subject | Retrovirus | en_US |
dc.subject | RNA analysis | en_US |
dc.subject | transactivation | en_US |
dc.subject | virus genome | en_US |
dc.subject | virus inhibition | en_US |
dc.title | Simultaneous RNA quantification of human and retroviral genomes reveals intact interferon signaling in HTLV-1-infected CD4+ T cell lines | en_US |
dc.type | info:eu-repo/semantics/article | |
dc.identifier.doi | https://doi.org/10.1186/1743-422X-9-171 | |
dc.subject.ocde | https://purl.org/pe-repo/ocde/ford#1.06.02 | |
dc.relation.issn | 1743-422X |
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